Lyme Disease is not Caused by a Microorganism
A spirochete bacteria does not cause Lyme disease. Ticks do not cause disease, they trigger something already underlying. People are bitten by ticks all the time without issue. They either cause disease or they don’t. Germ theorists often create another lie to cover this one up, that of “immunity”, another false concept. That lie was made to hide the truth and sell you the latest money-making vaccine. We do not have an immune system. That term came from a doctor at a symposium presenting information about our lymphatic system, an actual system of the body that is responsible for cleansing, tagging, and releasing wastes. Antibodies are non-specific. The testing racket produces false positives and negatives all the time. We are being lied to about how our bodies work and poisoned by the same people who then try to find new diseases to blame for the problems they cause.
What is Lyme then? Well, it is a system that has been poisoned and triggered into inflammation and release. Usually by vaccines, chronic antibiotic use, other heavy metals, microplastics, traumas, and/or nutritional deficiencies, as we see with literally all disease states. Presentations of symptoms will change based on the type of tissue that is involved, the hell-fire combination of chemical wastes at play, the damage to the genetic (protein-building) system by said chemicals, and the resources available to assist the release.
Giving more antibiotics may provide temporary relief as the microorganisms are active in trying to metabolize and remove the waste from the body and the symptoms are produced in an attempt to release said wastes but it is an illusion of cure. Your microorganisms are there to clean up the mess. Killing them off makes the symptoms retreat and repeated use will break the release mechanism. The future for that individual will look like nervous system diseases (MS, CF), dementia, Parkinsons, cancer, heart disease, liver disease, kidney disease, fertility issues, arthritis, osteoporosis, and reduction of longevity. The toxins are still there, just festering, leading to backup mechanisms by the brilliant body to be leaned on.
Improper treatment occurs due to false diagnosis as nutritional stores are not replenished and microbial support is not given. More bacteria need to be given, and we must not be killing anything at all. The germ theorist has a hankering for killing, completely misunderstanding their valuable role in nature, parasites included. If you are going to kill or chase away the microorganisms and parasites, then you’d better be ready to take over their job.
In Lyme disease, the common nutritional deficiencies witnessed are vitamin D3, B1, ALA (alpha lipoic acid), glutathione, magnesium, CoQ10, B12, zinc, and bacteria (probiotics). The body has used up these particular stores attempting to clean up the wastes and/or the damage that has been done prevents said nutrients from being used properly. There becomes a chronic deficiency of enzymes, as the bacteria have been so battered by incorrect treatments that they are not producing enough of them. Supplying the body with enzymes therefore becomes part of the treatment protocol. I suggest serrapeptase which is a systemic enzyme. Coffee enemas and urine therapy are also very important remedies to cleanse the liver, blood, and tissues and communicate bodily needs with the intelligence of water memory. Of course, DMSO can always be used to assist cleansing needs, repair tissue, control inflammation, and increase glutathione levels. An iTeraCare device would also help tremendously, as terahertz frequencies assist in detoxification and repair.
An individual on the edge of a purge only needs one event to trigger themselves into an attempted release. That could be a shock, a bite from any organism leading to inflammation, another poisoning from a pharmaceutical, or even improper eating. We see this with states of fibromyalgia and chronic fatigue, as well. These individuals are ticking time bombs and it only takes one trigger to set them off where the body now needs to go into a cleansing cycle that produces symptoms, symptoms which people judge as “negative” rather than understanding what the body is attempting to do. The medical cartel is standing by to continue to suppress those symptoms with drugs completely ignoring the root cause, primarily because they are the ones who caused it with their poisonous vaccines in the first place.
There is your Lyme disease, there is your COVID-19, there is your long COVID, there is your <insert any disease you want that is blamed on microorganisms and viruses.> When you come to the terrain understanding of illness, things become simpler, treatments become streamlined, and new options become available.
If you wish to heal from “Lyme” I suggest my Heavy Metals and Parasite Cleanse Course to clean up the terrain. Anyone with a chronic disease can benefit from this protocol.
Borrelia studies
Isolation of Borrelia burgdorferi sensu lato from blood of adult patients with borrelial lymphocytoma, Lyme neuroborreliosis, Lyme arthritis and acrodermatitis chronica atrophicans.
https://sci-hub.st/10.1007/s15010-010-0062-8
Results: “Borrelia burgdorferi sensu lato was isolated from the blood of 1/53 (1.9%) patients with borrelial lymphocytoma, 6/176 (3.4%) patients with Lyme neuroborreliosis, 1/13 (7.7%) patients with Lyme arthritis, and 3/ 200 (1.5%) patients with acrodermatitis chronica atrophicans.”
[…]
“At the time of the blood culture, erythema migrans was present in 4/11 (36.4%) borrelia blood culture-positive patients …”
Isolation of Borrelia Spirochetes from Patients in Texas
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC269164/
Results: “During this study, blood specimens from 100 patients were cultured for B. burgdorferi. We cultivated spirochetes from two (2%) specimens: one chilled whole blood and one frozen clot. A total of 31 skin specimens were placed in culture, resulting in 8 (26%) isolates, and spirochetes were observed in 1 of 7 (14%) cultured CSF specimens. Tissues from two autopsies (kidney, liver, spleen, lung, and lymph node) were submitted, and spirochetes were detected in the liver and spleen of one patient. Spirochetes were isolated from one of three (33%) joint fluids (knee) and from the one bone specimen that was submitted (wrist).”
Here’s a summary of the paper on the alleged causative agent of Lyme disease:
– Here is the first paper from 1982 describing experiments with the ‘isolated’ bacterium Borrelia burgdorferi: https://escholarship.org/content/qt9vj3t37b/qt9vj3t37b.pdf?t=p4g42c
– Only epidemiological evidence is referred to as causality, there is no direct evidence that or how the bacterium triggers the symptoms.
– The clinical symptoms are described as diffuse: an inflammatory disease that is initially noticeable through skin lesions. It was first recognized as a form of inflammatory arthritis in Lyme, Connecticut, although the clinical disease was also observed in other states.
– In order to prove the causality, experiments were then carried out with 8 rabbits. 30-40 ticks were taped to the shaved abdomen of each rabbit. That would be comparable to a person weighing 185lb (83 kg) having over 1,000 ticks stuck to their stomach. (after weight comparison).
– The bacterium was NEVER detected directly in the rabbits, only by means of antibody levels, which are not specific, as the authors themselves show, since the antibody tests always react, just in different dilutions.
– The antibody test used also reacts positively in people who do not suffer from the disease, only in lower concentrations.
– It is also important that these experiments were carried out without scientific controls. (e.g. rabbit ticks without bacteria, same procedure)
– A pure isolate of the bacterium was not obtained or used, so it is not possible to distinguish whether the clinical symptoms (skin inflammation) were caused by the bite wounds of the ticks or by the bacterium.
Willy Burgdorfer: Lyme disease (1996)
https://sci-hub.st/10.1016/s0736-4679(96)00143-6
”Laboratory diagnosis of Lyme disease remains problematic. Although Borrelia burgdorferi organisms have been identified in biopsy specimens of erythema migrans lesions (IS), such biopsy is not practical for emergency physicians and many other clinicians. Serologic testing is of very limited value in acute cases, inasmuch as IgM levels do not begin to rise for 2-4 weeks following the onset of infection (17). Immunological tests such as enzyme-linked immunosorbent assay (ELBA) and immunofluorescence assay suffer from poor reproducibility of positive and negative results (IO). Western immunoblotting, however, may be useful in identifying false-positive ELISA results (19). Consequently, currently, there is no standard laboratory test diagnostic for Lyme disease (12), and the diagnosis is best made on clinical and epidemiologic grounds.”
[…]
”Chronic Lyme disease may be overdiagnosed, often on the basis of false-positive serology …”
Recent Progress in Lyme Disease and Remaining Challenges
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8416313/
“The diagnosis of LD [lyme disease] can be a complex task for the provider because, outside of the erythema migrans (EM) lesion of early LD, diagnosis relies on non-specific clinical signs and symptoms that may or may not be supported by laboratory evidence.”
[…]
“LD is known as the “great imitator” because symptoms are varied and often overlap with common health complaints, sometimes making early diagnosis more difficult. … There are likely hundreds of health conditions with significant overlap with these non-specific signs and symptoms.”
Hollstrom E: Penicillin treatment of erythema chronicum migrans Afzelius
https://www.medicaljournals.se/acta/content_files/files/pdf/38/5/38285289.pdf
Thank you to a colleague for amassing Lyme research.
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